연구동향 분석

A Hyperactive Neutrophil Phenotype in Aggressive Periodontitis

Kap Yul Kim Min-Kyung Kim Yun Sik Choi Yong Cheol Kim Ah-Ram Jo In-Chul Rhyu Youngnim Choi 2012년

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' A Hyperactive Neutrophil Phenotype in Aggressive Periodontitis' 의 주제별 논문영향력
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  • 치과의학, 이비인후과학, 안과학
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' A Hyperactive Neutrophil Phenotype in Aggressive Periodontitis' 의 참고문헌

  • The novel applications of the quantitative analysis of neutrophil cell surface FcgammaRI (CD64) to the diagnosis of infectious and inflammatory diseases
    Nutila J [2010]
  • The Fcgamma receptor genotype as a risk factor for generalized early-onset periodontitis in Japanese patients
    Kobaynshi T [2000]
  • Systemic release of endotoxins induced by gentle mastication: association with periodontitis severity
    Geerts SO [2002]
  • Subcellular localization and dynamics of Mac-1 (alpha m beta 2) in human neutrophils
    Dimond MS [1993]
  • Simultaneous quantitative analysis of Fc gamma RI (CD64) and CR1 (CD35) on neutrophils in distinguishing between bacterial infections, viral infections, and inflammatory diseases
    Hohenthal U Simultaneous quantitative analysis of Fc gamma RI (CD64) and CR1 (CD35) on neutrophils in distinguishing between bacterial infections [2009]
  • Relevance of immunoglobulin G Fc receptor polymorphism to recurrence of adult periodontitis in Japanese patients
    Suzuki [1997]
  • Regulation of superoxide production in neutrophils: role of calcium influx
  • Redox regulation of Fcgamma receptor- mediated phagocytosis: implications for host defense and tissue injury
    Pricop L [2002]
  • Priming response to inflammatory mediators in hyperreactive peripheral neutrophils from adult periodontitis
    Gustaffsson [1997]
  • Peripheral PMN cells in juvenile periodontitis. Increased release of elastase and of oxygen radicals after stimulation with opsonized bacteria
    Asman B [1988]
  • Neutrophils, from marrow to microbes
    Borregard N Neutrophils [2010]
  • Neutrophil hyper-responsiveness in periodontitis
    Robert [2007]
  • NADPH oxidase (CYBA) and FcgammaR polymorphisms as risk factors for aggressive periodontitis: a case-control association study
    Nibali L [2006]
  • Micobial etiology of periodontitis
    Nishihara T [2004]
  • Memrane-bound proteinase 3 and PAR2 mediate phagocytosis of non-opsonized bacteria in human neutrophils
    Kim YC [2011]
  • Increased respiratory burst activity is associated with normal expression of IgG-Fc-receptors and complement receptors in peripheral neutrophils from patients with juvenile periodontitis
    Leino L [1994]
  • Increased release of free oxygen radicals from peripheral neutrophils in adult periodontitis after Fc gamma-receptor stimulation
    Asman B [1996]
  • Hyperactivity and reactivity of peripheral blood neutrophils in chronic periodontitis
    CooperPR [2007]
  • Hyper-reactive peripheral neutrophils in adult periodontitis: generation of chemiluminescence and intracellular hydrogen peroxide after in vitro priming and FcgammaRstimulation
  • Hyper-reactive PMNs in FcgammaRIIa 131 H/H genotype periodontitis patients
    Asman B [2007]
  • Human oral, gut, and plaque microbiota in patients with atherosclerosis
    Koren O Human oral [2011]
  • Human Fc receptors: critical targets in the treatment of autoimmune diseases and transplant rejections
    I van E [2006]
  • Hematological indices, inflammatory markers and neutrophil CD64 expression: comparative trends during experimental human endotoxemia
    Vandermeer M Hematological indices [2007]
  • Constitutionally hyperreactive neutrophils in periodontitis
  • Changes in subcellular localization and surface expression of L-selectin, alkaline phosphatase, and Mac-1 in human neutrophils during stimulation with inflammatory mediators
    Thomas Angelo Changes in subcellular localization and surface expression of L-selectin [1994]
  • Association of endotoxemia with carotid atherosclerosis and cardiovascular disease: prospective results from the Bruneck Study
  • Ability of oral bacteria to induce tissue-destructive molecules from human neutrophils
  • A hyperactive neutrophil phenotype in patients with refractory periodontitis
    Kuh [2007]