Particulate matter promotes cancer metastasis through increased HBEGF expression in macrophages
활용도 Analysis
논문 Analysis
연구자 Analysis
'
Particulate matter promotes cancer metastasis through increased HBEGF expression in macrophages' 의 참고문헌
p38 stabilizes snail by suppressing DYRK2-mediated phosphorylation that is required for GSK3β-βTrCP-induced Snail degradation
The mechanisms for lung cancer risk of PM(2.5): Induction of epithelial-mesenchymal transition and cancer stem cell properties in human nonsmall cell lung cancer cells
The innate and adaptive immune response induced by alveolar macrophages exposed to ambient particulate matter
The effects for PM2.5 exposure on non-smallcell lung cancer induced motility and proliferation
The aryl hydrocarbon receptor as a target of environmental stressors – implications for pollution mediated stress and inflammatory responses
The ERBB network : at last, cancer therapy meets systems biology
Targeting EMT in cancer : opportunities for pharmacological intervention
Soluble HB-EGF induces epithelial-to-mesenchymal transition in inner medullary collecting duct cells by upregulating Snail-2
Secondhand smoke-prevalent polycyclic aromatic hydrocarbon binary mixture-induced specific mitogenic and pro-inflammatory cell signaling events in lung epithelial cells
SOX2 promotes brain metastasis of breast cancer by upregulating the expression of FSCN1 and HBEGF
Regulatory mechanisms of the HB-EGF autocrine loop in inflammation, homeostasis, development and cancer
Phagocytosis of particulate air pollutants by human alveolar macrophages stimulates the bone marrow
Particulate matter induces cytokine expression in human bronchial epithelial cells
Particulate matter air pollution stimulates monocyte release from the bone marrow
PM2.5 exposure in the respiratory system induces distinct inflammatory signaling in the lung and the liver of mice
Outdoor particulate matter exposure and lung cancer : A systematic review and meta-analysis
Multiple G-protein-coupled receptor signals converge on the epidermal growth factor receptor to promote migration and invasion
Mechanisms of receptor tyrosine kinase activation in cancer
LIV-1 promotes prostate cancer epithelial-to-mesenchymal transition and metastasis through HB-EGF shedding and EGFR-mediated ERK signaling
Interactions between cancer cells and immune cells drive transitions to mesenchymal-like states in glioblastoma
Interaction of alveolar macrophages and airway epithelial cells following exposure to particulate matter produces mediators that stimulate the bone marrow
Inhaled particulate matter causes expression of nuclear factor(NF)-kappaB-related genes and oxidant-dependent NF-kappaB activation in vitro
Inflammation and cancer: back to Virchow?
Impact of air pollution on the burden of chronic respiratory diseases in China : time for urgent action
Heparin-binding epidermal growth factorlike growth factor promotes transcoelomic metastasis in ovarian cancer through epithelial-mesenchymal transition
Heparin-binding epidermal growth factor-like growth factor(HB-EGF)and proteolytic processing by a disintegrin and metalloproteinases(ADAM) : a regulator of several pathways
Heparin-binding epidermal growth factor (HB-EGF) drives EMT in patients with COPD: implications for disease pathogenesis and novel therapies
Heparin-binding EGF-like growth factor promotes epithelial-mesenchymal transition in human keratinocytes
HIF-1α, NOTCH1, ADAM12, and HB-EGF are overexpressed in mucoepidermoid carcinoma
HBEGF(+) macrophages in rheumatoid arthritis induce fibroblast invasiveness
HB-EGF is a promising therapeutic target for lung cancer with secondary mutation of EGFR(T790M)
Global estimates of mortality associated with long-term exposure to outdoor fine particulate matter
Global burden of 87 risk factors in 204 countries and territories, 1990-2019: a systematic analysis for the Global Burden of Disease Study 2019
Expression of heparin-binding epidermal growth factor-like growth factor in pancreatic adenocarcinoma
Expression and role of EGFR ligands induced in airway cells by PM2.5 and its components
Exposure to ultrafine particulate matter induces NF-κβ mediated epigenetic modifications
Epithelial-mesenchymal transitions in tumour progression .
Epithelial-mesenchymal transitions in development and disease
Epidermal growth factor receptor cooperates with signal transducer and activator of transcription 3 to induce epithelial-mesenchymal transition in cancer cells via up-regulation of TWIST gene expression
Epidermal growth factor receptor cell proliferation signaling pathways
E6-mediated activation of JNK drives EGFR signalling to promote proliferation and viral oncoprotein expression in cervical cancer
Downregulation of CHIP promotes ovarian cancer metastasis by inducing Snail-mediated epithelial-mesenchymal transition
Bacterial type III effector protein HopQ inhibits melanoma motility through autophagic degradation of vimentin
BHLHE40 confers a pro-survival and pro-metastatic phenotype to breast cancer cells by modulating HBEGF secretion
Aryl hydrocarbon receptor in association with RelA modulates IL-6 expression in non-smoking lung cancer
Aryl hydrocarbon receptor (AhR) agonists increase airway epithelial matrix metalloproteinase activity
Appearance of heparin-binding EGF-like growth factor in wound fluid as a response to injury
Alveolar macrophages drive hepatocellular carcinoma lung metastasis by generating leukotriene B(4)
Air pollution and lung cancer incidence in 17 European cohorts : prospective analyses from the European Study of Cohorts for Air Pollution Effects(ESCAPE)
Air pollutants, oxidative stress and human health
AhR signaling pathways and regulatory functions
Afatinib, an EGFR inhibitor, decreases EMT and tumorigenesis of Huh‑7 cells by regulating the ERK‑VEGF/MMP9 signaling pathway
A heparinbinding growth factor secreted by macrophage-like cells that is related to EGF
2,3,7,8-Tetrachlorodibenzo-p-dioxin-induced degradation of aryl hydrocarbon receptor (AhR) by the ubiquitin-proteasome pathway. Role of the transcription activaton and DNA binding of AhR
'
Particulate matter promotes cancer metastasis through increased HBEGF expression in macrophages'
의 유사주제(
