Transcriptional regulation and chromatin dynamics at DNA double-strand breaks
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Transcriptional regulation and chromatin dynamics at DNA double-strand breaks' 의 참고문헌
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WWP2 ubiquitylates RNA polymerase II for DNA-PK-dependent transcription arrest and repair at DNA breaks
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Understanding nucleotide excision repair and its roles in cancer and ageing
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Ubiquitin-binding protein RAP80 mediates BRCA1-dependent DNA damage response
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Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions
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Transcriptionally active chromatin recruits homologous recombination at DNA double-strand breaks
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Transcriptional elongation factor ENL phosphorylated by ATM recruits polycomb and switches offtranscription for DSB repair
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Transcription-Coupled DNA Double-Strand Break Repair : Active Genes Need Special Care
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The p400 ATPase regulates nucleosome stability and chromatin ubiquitination during DNA repair
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The chromatin remodeller RSF1 is essential for PLK1 deposition and function at mitotic kinetochores
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The chromatin remodeler RSF1 coordinates epigenetic marks for transcriptional repression and DSB repair
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The chromatin remodeler RSF1 controls centromeric histone modifications to coordinate chromosome segregation
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The biology of chromatin remodeling complexes
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The RSF1 histone-remodelling factor facilitates DNA double-strand break repair by recruiting centromeric and Fanconi Anaemia proteins
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The RIDDLE syndrome protein mediates a ubiquitindependent signaling cascade at sites of DNA damage
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The OTUD5-UBR5 complex regulates FACT-mediated transcription at damaged chromatin
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The DNA-damage response in human biology and disease
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The AAA-ATPase VCP/p97 promotes 53BP1 recruitment by removing L3MBTL1 from DNA double-strand breaks
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Spatiotemporal coordination of the RSF1-PLK1-Aurora B cascade establishes mitotic signaling platforms
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Screen identifies bromodomain protein ZMYND8 in chromatin recognition of transcription-associated DNA damage that promotes homologous recombination
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Screen identifies DYRK1B network as mediator of transcription repression on damaged chromatin
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Role of histone H2A ubiquitination in Polycomb silencing
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Rif1 prevents resection of DNA breaks and promotes immunoglobulin class switching
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Requirement for PBAF in transcriptional repression and repair at DNA breaks in actively transcribed regions of chromatin
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Repression of Transcription at DNA Breaks Requires Cohesin throughout Interphase and Prevents Genome Instability
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Remodeling and spacing factor 1(RSF1)deposits centromere proteins at DNA double-strand breaks to promote non-homologous end-joining
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Remodeling and spacing factor 1(RSF1) : a rising star in DNA repair
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ROS-induced R loops trigger a transcription-coupled but BRCA1/2-independent homologous recombination pathway through CSB
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RNF168 binds and amplifies ubiquitin conjugates on damaged chromosomes to allow accumulation of repair proteins
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RIF1 is essential for 53BP1-dependent nonhomologous end joining and suppression of DNA double-strand break resection
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RAP80 targets BRCA1 to specific ubiquitin structures at DNA damage sites
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R-loops as Janus-faced modulators of DNA repair
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Proteome-wide identification of poly(ADP-ribose) binding proteins and poly(ADP-ribose)-associated protein complexes
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Proteome-wide identification of poly(ADP-Ribosyl)ation targets in different genotoxic stress responses
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Preferential repair of DNA double-strand break at the active gene in vivo
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Poly(ADP-ribosyl)ation links the chromatin remodeler SMARCA5/SNF2H to RNF168-dependent DNA damage signaling
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Poly(ADP-ribose)-dependent regulation of DNA repair by the chromatin remodeling enzyme ALC1
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PARP1-dependent recruitment of the FBXL10-RNF68-RNF2 ubiquitin ligase to sites of DNA damage controls H2A
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PARP1-dependent kinetics of recruitment of MRE11 and NBS1proteins to multiple DNA damage sites
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NELF-E is recruited to DNA double-strand break sites to promote transcriptional repression and repair
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Megabase chromatin domains involved in DNA double-strand breaks in vivo
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Mechanisms of action and regulation of ATP-dependent chromatin-remodelling complexes
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Mdc1 couples DNA double-strand break recognition by Nbs1 with its H2AX-dependent chromatin retention
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MDC1 maintains genomic stability by participating in the amplification of ATM-dependent DNA damage signals
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MDC1 directly binds phosphorylated histone H2AX to regulate cellular responses to DNA double-strand breaks
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Interaction of BARD1 and HP1 Is Required for BRCA1 Retention at Sites of DNA Damage
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Human Rad52 Promotes XPG-Mediated R-loop Processing to Initiate Transcription-Associated Homologous Recombination Repair
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Histone ubiquitination in the DNA damage response
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Histone demethylase KDM5A regulates the ZMYND8-NuRD chromatin remodeler to promote DNA repair
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Histone H2AK119 Mono-Ubiquitination Is Essential for Polycomb-Mediated Transcriptional Repression
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Histone H2A.Z controls a critical chromatin remodeling step required for DNA double-strand break repair
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Histone H1 couples initiation and amplification of ubiquitin signalling after DNA damage
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H4K20me0 recognition by BRCA1-BARD1 directs homologous recombination to sister chromatids
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Genome-wide mapping of long-range contacts unveils clustering of DNA double-strand breaks at damaged active genes
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Genome Regulation by Polycomb and Trithorax : 70 Years and Counting
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Functional transcription promoters at DNA double-strand breaks mediate RNA-driven phase separation of damage-response factors
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Express or repress? The transcriptional dilemma of damaged chromatin
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De novo phosphorylation of H2AX by WSTF regulates transcriptioncoupled homologous recombination repair
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DNAPKcs-dependent arrest of RNA polymerase II transcription in the presence of DNA breaks
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DNA doublestranded breaks induce histone H2AX phosphorylation on serine 139
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DNA double strand break repair pathway choice: a chromatin based decision?
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DNA damage-induced transcription stress triggers the genomewide degradation of promoter-bound Pol II
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DNA damage during the G0/G1 phase triggers RNA-templated, Cockayne syndrome B-dependent homologous recombination
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Comprehensive Mapping of Histone Modifications at DNA Double-Strand Breaks Deciphers Repair Pathway Chromatin Signatures
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Chromatin-remodeling factor, RSF1, controls p53-mediated transcription in apoptosis upon DNA strand breaks
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Chromatin remodeling at DNA double-strand breaks
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Cell cycle-dependent complex formation of BRCA1.CtIP.MRN is important for DNA double-strand break repair
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Cancer biology and NuRD : a multifaceted chromatin remodelling complex
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CDYL1 fosters double-strand break-induced transcription silencing and promotes homology-directed repair
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BMI1-UBR5 axis regulates transcriptional repression at damaged chromatin
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Abraxas and RAP80 form a BRCA1 protein complex required for the DNA damage response
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ATP-dependent chromatin remodeling in the DNA-damage response
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ATM-dependent chromatin remodeler Rsf-1 facilitates DNA damage checkpoints and homologous recombination repair
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ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks
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ATM-Dependent Recruitment of BRD7 is required for Transcriptional Repression and DNA Repair at DNA Breaks Flanking Transcriptional Active Regions
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APC/C(Cdh1)controls CtIP stability during the cell cycle and in response to DNA damage
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A damaged genome’s transcriptional landscape through multilayered expression profiling around in situ-mapped DNA double-strand breaks
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A chromatin localization screen reveals poly (ADP ribose)-regulated recruitment of the repressive polycomb and NuRD complexes to sites of DNA damage
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A cell cycle-dependent regulatory circuit composed of 53BP1-RIF1 and BRCA1-CtIP controls DNA repair pathway choice
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A Snapshot on the Cis Chromatin Response to DNA Double-Strand Breaks
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A POLD3/BLM dependent pathway handles DSBs in transcribed chromatin upon excessive RNA : DNA hybrid accumulation
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53BP1regulates DSB repair using Rif1 to control 5’ end resection
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53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers
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53BP1 is a reader of the DNA-damage-induced H2A Lys 15 ubiquitin mark
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53BP1 inhibits homologous recombination in Brca1-deficient cells by blocking resection of DNA breaks
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Transcriptional regulation and chromatin dynamics at DNA double-strand breaks'
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