3, 5, 30-Triiodothyroacetic acid (TRIAC) is an anti-inflammatory drug that targets toll-like receptor 2
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3, 5, 30-Triiodothyroacetic acid (TRIAC) is an anti-inflammatory drug that targets toll-like receptor 2' 의 참고문헌
Triiodothyroacetic acid (TRIAC) potentiation of antidepressant-induced reversal of learned helplessness in rats
Toll-like receptors 1 and 6 are involved in TLR2-mediated macrophage activation by hepatitis C virus core and NS3 proteins
Toll-like receptor signaling pathways
Toll-like receptor signaling
Thyroid hormones derivatives reduce proliferation and induce cell death and DNA damage in ovarian cancer
Thyroid hormone regulation of metabolism
Thyroid hormone analogue stimulates keratinocyte proliferation but inhibits cell differentiation in epidermis
The relationship between the thyroid gland and the liver
The concanavalin A model of acute hepatitis in mice
Tetrac can replace thyroid hormone during brain development in mouse mutants deficient in the thyroid hormone transporter mct8
Sophocarpine protects mice from ConA-Induced hepatitis via inhibition of the IFN-Gamma/STAT1 Pathway
Serum free triiodothyronine (T3) to free thyroxine (T4) ratio in treated central hypothyroidism compared with primary hypothyroidism and euthyroidism
Resveratrol pretreatment attenuates concanavalin a-induced hepatitis through reverse of aberration in the immune response and regenerative capacity in aged mice
Regulation of Toll-like receptor-2 expression in chronic hepatitis B by the precore protein
Quercetin protects mice from ConA-induced hepatitis by inhibiting HMGB1-TLR expression and down-regulating the nuclear factor kappa B pathway
Protective effects of soyasapogenol A on liver injury mediated by immune response in a concanavalin A-induced hepatitis model
Protective effects of astaxanthin on ConA-induced autoimmune hepatitis by the JNK/p-JNK pathway-mediated inhibition of autophagy and apoptosis
Protective effect of ellagic acid on concanavalin A-induced hepatitis via toll-like receptor and mitogen-activated protein kinase/nuclear factor kappaB signaling pathways
Phosphoinositide 3-kinase gamma inhibitor ameliorates concanavalin A-induced hepatic injury in mice
Modulation of rat liver mitochondrial antioxidant defence system by thyroid hormone
Interleukin-11 reduces T-cell-dependent experimental liver injury in mice
Interaction of plant lectins with purified human lymphocyte populations: binding characteristics and kinetics of proliferation
How we detect microbes and respond to them: the Toll-like receptors and their transducers
Higher thyroid-stimulating hormone, triiodothyronine and thyroxine values are associated with better outcome in acute liver failure
Emodin protects against concanavalin A-induced hepatitis in mice through inhibiting activation of the p38 MAPK-NF-kappaB signaling pathway
Drug repositioning: identifying and developing new uses for existing drugs
Depolarization and increased conductance precede superoxide release by concanavalin A-stimulated rat alveolar macrophages
Concanavalin A induced expression of Toll-like receptors in murine peritoneal macrophages in vitro
Clinical associations between thyroid and liver diseases
Chronic hepatitis C virus infection and neurological and psychiatric disorders: an overview
Blocking TLR2 in vivo attenuates experimental hepatitis induced by concanavalin A in mice
Augmentation of Valpha14 NKT cell-mediated cytotoxicity by interleukin 4 in an autocrine mechanism resulting in the development of concanavalin A-induced hepatitis
A toll-like receptor that prevents infection by uropathogenic bacteria
A simple practice guide for dose conversion between animals and human
A T cell-dependent experimental liver injury in mice inducible by concanavalin A
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3, 5, 30-Triiodothyroacetic acid (TRIAC) is an anti-inflammatory drug that targets toll-like receptor 2'
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