Recent Topics in Fibrodysplasia Ossificans Progressiva
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Recent Topics in Fibrodysplasia Ossificans Progressiva' 의 참고문헌
Two tissue-resident progenitor lineages drive distinct phenotypes of heterotopic ossification
Trichocyalides A and B, new inhibitors of alkaline phosphatase activity in bone morphogenetic protein-stimulated myoblasts, produced by Trichoderma sp. FKI-5513
The natural history of heterotopic ossification in patients who have fibrodysplasia ossificans progressiva. A study of forty-four patients
The natural history of flare-ups in fibrodysplasia ossificans progressiva (FOP): a comprehensive global assessment
The immunophilin FKBP12 functions as a common inhibitor of the TGF beta family type I receptors
The fibrodysplasia ossificans progressiva R206H ACVR1 mutation activates BMP-independent chondrogenesis and zebrafish embryo ventralization
The expansion of heterotopic bone in fibrodysplasia ossificans progressiva is activin a-dependent
Synthesis and structure-activity relationships of a novel and selective bone morphogenetic protein receptor (BMP) inhibitor derived from the pyrazolo[1.5-a]pyrimidine scaffold of dorsomorphin: the discovery of ML347 as an ALK2 versus ALK3 selective MLPCN probe
Smad9 is a new type of transcriptional regulator in bone morphogenetic protein signaling
Signal transduction by the TGF-beta superfamily
Scopranones with two atypical scooplike moieties produced by streptomyces sp. BYK-11038
Restoration of normal BMP signaling levels and osteogenic differentiation in FOP mesenchymal progenitor cells by mutant allele-specific targeting
Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-<TEX>$\gamma$</TEX> agonists
Palovarotene inhibits heterotopic ossification and maintains limb mobility and growth in mice with the human ACVR1(R206H) fibrodysplasia ossificans progressiva (FOP) mutation
Overexpression of an osteogenic morphogen in fibrodysplasia ossificans progressiva
Neofunction of ACVR1 in fibrodysplasia ossificans progressiva
Mutant activin-like kinase 2 in fibrodysplasia ossificans progressiva are activated via T203 by BMP type II receptors
Multipotent progenitors resident in the skeletal muscle interstitium exhibit robust BMP-dependent osteogenic activity and mediate heterotopic ossification
Mechanism of activation of the TGF-beta receptor
Lumbar puncture and surgical intervention in a child with undiagnosed fibrodysplasia ossificans progressiva
Linkage exclusion and mutational analysis of the noggin gene in patients with fibrodysplasia ossificans progressiva (FOP)
Inhibition of ectopic bone formation by a selective retinoic acid receptor alpha-agonist: a new therapy for heterotopic ossification?
In vivo structure-activity relationship study of dorsomorphin analogues identifies selective VEGF and BMP inhibitors
Identification of progenitor cells that contribute to heterotopic skeletogenesis
Identification of a novel bone morphogenetic protein (BMP)-inducible transcript, BMP-inducible transcript-1, by utilizing the conserved BMP-responsive elements in the Id genes
Identification of a BMP-responsive element in Id1, the gene for inhibition of myogenesis
Iatrogenic harm caused by diagnostic errors in fibrodysplasia ossificans progressiva
Heterotopic bone formation induced by bone morphogenetic protein signaling: fibrodysplasia ossificans progressiva
Fungal pyrrolidine-containing metabolites inhibit alkaline phosphatase activity in bone morphogenetic protein-stimulated myoblastoma cells
Fibrodysplasia ossificans progressiva: mechanisms and models of skeletal metamorphosis
Fibrodysplasia ossificans progressiva, a heritable disorder of severe heterotopic ossification, maps to human chromosome 4q27-31
Fibrodysplasia Ossificans Progressiva Consortium. Localization of the gene for fibrodysplasia ossificans progressiva (FOP) to chromosome 17q21-22
Effects of FKBP12 and type II BMP receptors on signal transduction by ALK2 activating mutations associated with genetic disorders
Early diagnosis of fibrodysplasia ossificans progressiva
Dual roles of Smad proteins in the conversion from myoblasts to osteoblastic cells by bone morphogenetic proteins
Dorsomorphin inhibits BMP signals required for embryogenesis and iron metabolism
Disease-causing allele-specific silencing against the ALK2 mutants, R206H and G356D, in fibrodysplasia ossificans progressiva
Development of an ALK2-biased BMP type I receptor kinase inhibitor
Deformity of the great toe in fibrodysplasia ossificans progressiva
Conversion of vascular endothelial cells into multipotent stem-like cells
Constitutively activated ALK2 and increased SMAD1/5 cooperatively induce bone morphogenetic protein signaling in fibrodysplasia ossificans progressiva
Clinically applicable antianginal agents suppress osteoblastic transformation of myogenic cells and heterotopic ossifications in mice
Classic and atypical fibrodysplasia ossificans progressiva (FOP) phenotypes are caused by mutations in the bone morphogenetic protein (BMP) type I receptor ACVR1
Bone: formation by autoinduction
BMP type I receptor inhibition reduces heterotopic [corrected] ossification
Antisense-oligonucleotide mediated exon skipping in activin-receptor-like kinase 2: inhibiting the receptor that is overactive in fibrodysplasia ossificans progressiva
An Acvr1 R206H knock-in mouse has fibrodysplasia ossificans progressiva
Activin-dependent signaling in fibro/adipogenic progenitors causes fibrodysplasia ossificans progressiva
Activin-A enhances mTOR signaling to promote aberrant chondrogenesis in fibrodysplasia ossificans progressiva
ACVR1R206H receptor mutation causes fibrodysplasia ossificans progressiva by imparting responsiveness to activin A
A unique mutation of ALK2, G356D, found in a patient with fibrodysplasia ossificans progressiva is a moderately activated BMP type I receptor
A recurrent mutation in the BMP type I receptor ACVR1 causes inherited and sporadic fibrodysplasia ossificans progressiva
A novel ACVR1 mutation in the glycine/serine-rich domain found in the most benign case of a fibrodysplasia ossificans progressiva variant reported to date
A new class of small molecule inhibitor of BMP signaling
A door opens for fibrodysplasia ossificans progressiva
5-Prenyltryptophol, a new inhibitor of bone morphogenetic protein-induced alkaline phosphatase expression in myoblasts, produced by Streptomyces colinus subsp. Albescens HEK608
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Recent Topics in Fibrodysplasia Ossificans Progressiva'
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