HepG2 인체 간암세포의 ROS 생성 및 ERK/Akt 신호전달 경로 조절을 통한 sanguinarine의 apoptosis 유도

논문상세정보
' HepG2 인체 간암세포의 ROS 생성 및 ERK/Akt 신호전달 경로 조절을 통한 sanguinarine의 apoptosis 유도' 의 주제별 논문영향력
논문영향력 선정 방법
논문영향력 요약
주제
  • 생물과학
  • apoptosis
  • extracellularsignal-regulatedkinase(erk)
  • pi3k/akt
  • reactive oxygen species (ros)
  • sanguinarine
동일주제 총논문수 논문피인용 총횟수 주제별 논문영향력의 평균
1,295 0

0.0%

' HepG2 인체 간암세포의 ROS 생성 및 ERK/Akt 신호전달 경로 조절을 통한 sanguinarine의 apoptosis 유도' 의 참고문헌

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